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Health Secretary Robert F. Kennedy Jr. reportedly plans to acknowledge an acetaminophen-autism link, sending Tylenol maker Kenvue’s stock plummeting. Is scientist William Parker’s hypothesis correct, that acetaminophen (Tylenol) causes autism in some children? If so, is that solely or partly because its neurotoxicity aggravates the neurologic injury from mercury injection done by the Extraterrestrial Alliance?
ClosedNicola asked 1 week ago • 
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A recent study showed that nine pharmaceuticals commonly used in residential aged care significantly enhanced bacterial resistance to antibiotics, adding to the growing worldwide problem of declining antibiotic effectiveness, and the attendant morbidity and mortality. These included Advil, Tylenol, diclofenac (an anti-inflammatory to treat arthritis), furosemide (for high blood pressure), metformin (for high sugar levels linked to diabetes), atorvastatin (to help lower cholesterol and fats in the blood), tramadol (a stronger pain medication post-surgery), temazepam (used to treat sleeping problems), and pseudoephedrine (a decongestant). Are these or other drugs also causing lowered immune defenses against chronic viruses, which you have confirmed are the cause of 85% of chronic diseases? Hanbiao Chen, Sylvia A. Sapula, John Turnidge, Henrietta Venter. The effect of commonly used non-antibiotic medications on antimicrobial resistance development in Escherichia coli. npj Antimicrobials and Resistance, 2025; 3 (1) DOI: 10.1038/s44259-025-00144-w.
ClosedNicola asked 1 week ago • 
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Was the research, including the clinical data, for the paper we asked you about, “Acute inflammatory response via neutrophil activation protects against development of chronic pain,” valid and not corrupted by the interlopers to malign seemingly useful common painkillers? Given your warnings that clinical research cannot always be trusted, we just want to be sure. What is most important for us to know?
ClosedNicola asked 1 week ago • 
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The phenomena seem quite striking which are reported in the paper, “Acute inflammatory response via neutrophil activation protects against development of chronic pain,” by Marc Parisien et al. Sci Transl Med. 2022 May 11; 14(644). These authors showed in animal models that painkillers like corticosteroids and NSAIDs, but not analgesics like morphine or lidocaine which lack anti-inflammatory effects, end up prolonging pain, because without neutrophil activation being allowed to happen as part of an immune response, acute pain may become chronic. A clinical study confirmed that among patients whose lower back pain had become chronic, it was exclusively those who had been treated with NSAIDS who went on to develop neuropathy. Was this a valid study with broad implications about the unexpected downside of anti-inflammatory medication for moderating pain? What is Creator’s perspective?
ClosedNicola asked 1 week ago • 
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I shudder when I consider I spent years striving to develop inhibitors of neutrophil activation as therapeutics for autoimmune disease. Was that not only misguided, but a sinister consequence of interloper manipulation to encourage the field of immunopharmacology, knowing it could make things worse? What can Creator tell us about the medical implications for using anti-inflammatories?
ClosedNicola asked 1 week ago • 
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Acetaminophen (paracetamol) also marketed as Tylenol, is one of the most widely-used drugs by pregnant women and children and studies have mounted showing a link between prenatal or early childhood exposure and neurodevelopmental disorders, like autism, attention-deficit hyperactivity disorder (ADHD), language delays, behavioral problems, and lower IQ. Are those studies accurate and valid, or has there been interloper interference to create another cover story to mask their deliberate toxic manipulations of the brains of infants?
ClosedNicola asked 1 week ago • 
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There is a description circulating on the Internet claiming that the polio epidemic was misinformation based on assuming the polio virus was causing paralysis when it was actually a dangerous upsurge in spraying DDT indiscriminately, which is neurotoxic. What is the divine truth here?
ClosedNicola asked 2 months ago • 
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A number of research studies have been called into question because of poor reproducibility when an attempt to replicate their findings failed. A “replication crisis” has been noted for psychology, sociology, education, economics, and medical research. For example, a detailed inquiry was mounted to replicate 193 experiments from 53 top cancer papers published from 2010-2012. Only a quarter of the original findings were able to be reproduced. Moreover, even for effects that were reproduced, the magnitude averaged 85% less than observed in the original investigations. This is concerning because such experiments are crucial for deciding what to pursue for drug development and when to proceed to clinical trials. Unreliable results not only waste money but expose patients to unnecessary risks. While there are many understandable reasons why results can vary, what can Creator tell us about hidden reasons for such poor reproducibility and their impact on the reliability of scientific inquiry?
ClosedNicola asked 2 months ago • 
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A recent post in Neuroscience News highlighted a new study that reveals a possible link between common herpesvirus infections and the early biological changes that precede dementia. “The strongest viral associations were seen for Epstein-Barr virus and HHV6 (human herpesvirus 6)…These findings support the hypothesis that chronic viral infections may accelerate neurodegeneration and raise dementia risk independently of genetic factors.” (“Human herpes viruses are associated with steeper age-dependent increases of serum biomarkers for dementia in cognitively unimpaired women” by Lisa M. James et al. Scientific Reports). Is this a valid clue supporting the viral causation of dementia?
ClosedNicola asked 2 months ago • 
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Arguably, the biggest champion over the years for cancer as a metabolic disease and largely ignored by medical opinion leaders, has been Dr. Thomas Seyfried who has promoted ketogenic diets as an adjunct to conventional treatment. A state of ketosis lowers blood sugar which tumor cells need, and elevates ketone bodies which normal cells can use for energy but tumor cells can’t. But even he seems blind to the clues about viral causation. In a 2014 study in Cancer Letters, he augments his theory of the central role of metabolism in glioblastoma in describing how 90% of brain tumor cells are infected by cytomegalovirus, “which is a supercharger for allowing tumor cells to use glucose and glutamine.” That biochemical pathway allows energy for tumors to grow despite inadequate oxygenation. But he doesn’t ask, why are viruses there to begin with and what other role might they play? Is this an example of mind control suppression to limit scientific progress?
ClosedNicola asked 2 months ago • 
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I am concerned about recommending people take 12 mg of ivermectin daily for 3 weeks (along with hydroxychloroquine). Was I over-influenced by having that interchange with [Name Withheld] about “his protocol” to believe that giving both compounds would effect a viral cleanout? Was this a lesson to me about being more thorough, knowing as I investigated the literature about appropriate doses of ivermectin I would see the pharmacokinetic data, revealing the inability of oral administration to reach antiviral concentrations? What is most important for me to know?
ClosedNicola asked 2 months ago • 
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Recommending use of ivermectin in a way that is not even scientifically supportable seems like a big risk, even if you say it will work. What am I missing? How can high protein binding not be a hindrance to achieving a therapeutic exposure to free drug? Is there a scientific phenomenon here not yet scientifically understood or appreciated, at least not be me? Can you give us a tutorial to explain your continued endorsement and value of ivermectin, in addition to hydroxychloroquine which has demonstrated in vivo efficacy as an antiviral?
ClosedNicola asked 2 months ago • 
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In a paper on the pharmacokinetics of ivermectin, they report that even at a very high dose, free plasma levels never get close to the concentration needed, shown by in vitro studies, to demonstrate antiviral activity because ivermectin is 93% bound to plasma proteins. Yet, you have reassured us that taking an oral dose of 12 mg daily for 3 weeks would be effective in many cases for getting a profound, if not total, eradication of susceptible chronic viruses. Can you explain how that is possible?
ClosedNicola asked 2 months ago • 
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Does cod liver oil contain significant antiviral activity, enough to diminish viral disease symptoms or even cure chronic viral maladies?
ClosedNicola asked 3 months ago • 
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You told us that the supplement, Pineal Guardian X was an effective cognitive enhancer and neuroprotectant, despite its unstated and likely modest concentration of its ingredients. You said you could help us resolve the mystery here after we do some research on what the supplement contains. What I have seen is that 8 of its 9 ingredients have been confirmed by scientific studies to possess antiviral activity, at least at high concentrations, while the manufacturer only describes other varied properties of these phytonutrients. Is there something being missed by researchers? Does one or more substance have much more potent activity on certain viruses than yet appreciated? Is there an unexpected synergistic effect of this particular combination of what may be only modest and weak antivirals? Or is there some other property making the supplement beneficial? What is most important for us to know?
ClosedNicola asked 3 months ago • 
104 views0 answers0 votes
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