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Health Secretary Robert F. Kennedy Jr. reportedly plans to acknowledge an acetaminophen-autism link, sending Tylenol maker Kenvue’s stock plummeting. Is scientist William Parker’s hypothesis correct, that acetaminophen (Tylenol) causes autism in some children? If so, is that solely or partly because its neurotoxicity aggravates the neurologic injury from mercury injection done by the Extraterrestrial Alliance?
ClosedNicola asked 1 month ago • 
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A recent study showed that nine pharmaceuticals commonly used in residential aged care significantly enhanced bacterial resistance to antibiotics, adding to the growing worldwide problem of declining antibiotic effectiveness, and the attendant morbidity and mortality. These included Advil, Tylenol, diclofenac (an anti-inflammatory to treat arthritis), furosemide (for high blood pressure), metformin (for high sugar levels linked to diabetes), atorvastatin (to help lower cholesterol and fats in the blood), tramadol (a stronger pain medication post-surgery), temazepam (used to treat sleeping problems), and pseudoephedrine (a decongestant). Are these or other drugs also causing lowered immune defenses against chronic viruses, which you have confirmed are the cause of 85% of chronic diseases? Hanbiao Chen, Sylvia A. Sapula, John Turnidge, Henrietta Venter. The effect of commonly used non-antibiotic medications on antimicrobial resistance development in Escherichia coli. npj Antimicrobials and Resistance, 2025; 3 (1) DOI: 10.1038/s44259-025-00144-w.
ClosedNicola asked 1 month ago • 
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Was the research, including the clinical data, for the paper we asked you about, “Acute inflammatory response via neutrophil activation protects against development of chronic pain,” valid and not corrupted by the interlopers to malign seemingly useful common painkillers? Given your warnings that clinical research cannot always be trusted, we just want to be sure. What is most important for us to know?
ClosedNicola asked 1 month ago • 
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The phenomena seem quite striking which are reported in the paper, “Acute inflammatory response via neutrophil activation protects against development of chronic pain,” by Marc Parisien et al. Sci Transl Med. 2022 May 11; 14(644). These authors showed in animal models that painkillers like corticosteroids and NSAIDs, but not analgesics like morphine or lidocaine which lack anti-inflammatory effects, end up prolonging pain, because without neutrophil activation being allowed to happen as part of an immune response, acute pain may become chronic. A clinical study confirmed that among patients whose lower back pain had become chronic, it was exclusively those who had been treated with NSAIDS who went on to develop neuropathy. Was this a valid study with broad implications about the unexpected downside of anti-inflammatory medication for moderating pain? What is Creator’s perspective?
ClosedNicola asked 1 month ago • 
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I shudder when I consider I spent years striving to develop inhibitors of neutrophil activation as therapeutics for autoimmune disease. Was that not only misguided, but a sinister consequence of interloper manipulation to encourage the field of immunopharmacology, knowing it could make things worse? What can Creator tell us about the medical implications for using anti-inflammatories?
ClosedNicola asked 1 month ago • 
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Acetaminophen (paracetamol) also marketed as Tylenol, is one of the most widely-used drugs by pregnant women and children and studies have mounted showing a link between prenatal or early childhood exposure and neurodevelopmental disorders, like autism, attention-deficit hyperactivity disorder (ADHD), language delays, behavioral problems, and lower IQ. Are those studies accurate and valid, or has there been interloper interference to create another cover story to mask their deliberate toxic manipulations of the brains of infants?
ClosedNicola asked 1 month ago • 
52 views0 answers0 votes
A number of research studies have been called into question because of poor reproducibility when an attempt to replicate their findings failed. A “replication crisis” has been noted for psychology, sociology, education, economics, and medical research. For example, a detailed inquiry was mounted to replicate 193 experiments from 53 top cancer papers published from 2010-2012. Only a quarter of the original findings were able to be reproduced. Moreover, even for effects that were reproduced, the magnitude averaged 85% less than observed in the original investigations. This is concerning because such experiments are crucial for deciding what to pursue for drug development and when to proceed to clinical trials. Unreliable results not only waste money but expose patients to unnecessary risks. While there are many understandable reasons why results can vary, what can Creator tell us about hidden reasons for such poor reproducibility and their impact on the reliability of scientific inquiry?
ClosedNicola asked 3 months ago • 
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A $100 million clinical study (ISCHEMIA) followed more than 5,000 men and women from 37 countries with severe but stable heart disease to assess the value of procedures to help their coronary artery disease. Was the ISCHEMIA clinical trial compromised by the interlopers to discourage use of stents and coronary bypass procedures?
ClosedNicola asked 4 years ago • 
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There have been prominent announcements from the FDA about the inadvisability and dangers of people using ivermectin to treat COVID-19. Caution about taking pill formulations intended for large animals by humans is understandable, but this seems to have the same feel as the trashing of hydroxychloroquine early and often by the medical establishment exaggeration of possible side effects and even alteration of the data in clinical trials to make it falsely look both ineffective and highly toxic. Is that planned or underway to deliberately undercut further studies of ivermectin?
ClosedNicola asked 5 years ago • 
583 views0 answers0 votes
Will further clinical studies of fluvoxamine in patients with COVID-19 be suppressed by the US medical establishment as they have done for hydroxychloroquine?
ClosedNicola asked 5 years ago • 
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In a preliminary study of adult outpatients with symptomatic COVID-19, patients treated with fluvoxamine, compared with placebo, had a lower likelihood of clinical deterioration. There was clinical deterioration in 0 of 80 patients in the fluvoxamine group and in 6 of 72 patients in the placebo group (absolute difference, 8.7% [95% CI, 1.8%-16.4%] from survival analysis; log-rank P = .009). The fluvoxamine group had 1 serious adverse event and 11 other adverse events, whereas the placebo group had 6 serious adverse events and 12 other adverse events. Is this a worthwhile agent for COVID-19 management? How would it compare with hydroxychloroquine or ivermectin?
ClosedNicola asked 5 years ago • 
543 views0 answers0 votes
An osteopathic physician and frequent spokesperson on the Internet says there are at least 10 ways that the SARS-CoV-2 mRNA vaccines can cause serious side effects in human recipients. Is this the case, or an exaggeration? Why is she beating this drum?
ClosedNicola asked 5 years ago • 
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Is there a danger that clinical trials of COVID-19 vaccines will be interfered with by the Extraterrestrial Alliance to show lack of effectiveness, even if the approach can work?
ClosedNicola asked 5 years ago • 
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Is there a likelihood the Extraterrestrial Alliance will rig a vaccine trial to falsely show effectiveness, so it will be embraced by the masses, but useless, except to deliver locater chips?
ClosedNicola asked 5 years ago • 
501 views0 answers0 votes
Is the information on chlorine dioxide effectiveness for curing malaria accurate, that it was 100% effective, as described by Dr. Andreas Ludwig Kalcker?
ClosedNicola asked 5 years ago • 
563 views0 answers0 votes
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